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Receptor-Interacting Protein 3/Calmodulin-Dependent Kinase II/Proline-Rich Tyrosine Kinase 2 Pathway is Involved in Programmed Cell Death in a Mouse Model of Brain Ischaemic Stroke

Wang B, Ma L, Liu L, et al. Receptor-Interacting Protein 3/Calmodulin-Dependent Kinase II/Proline-Rich Tyrosine Kinase 2 Pathway is Involved in Programmed Cell Death in a Mouse Model of Brain Ischaemic Stroke. Neuroscience. 2022;506:14-28. doi:10.1016/j.neuroscience.2022.09.009

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SARS-CoV-2 evolves to reduce but not abolish neutralizing action

Zhang Y, Ndzouboukou JB, Lin X, et al. SARS-CoV-2 evolves to reduce but not abolish neutralizing action. J Med Virol. 2023;95(1):e28207. doi:10.1002/jmv.28207

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SARS-CoV-2 evolves to reduce but not abolish neutralizing action

Zhang Y, Ndzouboukou JB, Lin X, et al. SARS-CoV-2 evolves to reduce but not abolish neutralizing action. J Med Virol. 2023;95(1):e28207. doi:10.1002/jmv.28207

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M6 A Modifier-Mediated Methylation Characterized by Diverse Prognosis, Tumor Microenvironment, and Immunother

Liu, F., et al. (2022). M6A modifier‑mediated methylation characterized by diverse prognosis, tumor microenvironment, and immunotherapy response in hepatocellular carcinoma. [Journal unspecified].

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M6 A Modifier-Mediated Methylation Characterized by Diverse Prognosis, Tumor Microenvironment, and Immunother

Liu, F., et al. (2022). M6A modifier‑mediated methylation characterized by diverse prognosis, tumor microenvironment, and immunotherapy response in hepatocellular carcinoma. [Journal unspecified].

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SEMA4D/PlexinB1 promotes AML progression via activation of PI3K/Akt signaling

Liu L, Yang L, Liu X, et al. SEMA4D/PlexinB1 promotes AML progression via activation of PI3K/Akt signaling. J Transl Med. 2022;20(1):304. Published 2022 Jul 6. doi:10.1186/s12967-022-03500-w

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SEMA4D/PlexinB1 promotes AML progression via activation of PI3K/Akt signaling

Liu L, Yang L, Liu X, et al. SEMA4D/PlexinB1 promotes AML progression via activation of PI3K/Akt signaling. J Transl Med. 2022;20(1):304. Published 2022 Jul 6. doi:10.1186/s12967-022-03500-w

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Evolutionarily conserved odorant-binding proteins participate in establishing tritrophic interactions

Yang, R., Li, D., Yi, S., & Wang, M. (2022). Evolutionarily conserved odorant-binding proteins participate in establishing tritrophic interactions. iScience, 25(7), Article 104664.

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Evolutionarily conserved odorant-binding proteins participate in establishing tritrophic interactions

Yang, R., Li, D., Yi, S., & Wang, M. (2022). Evolutionarily conserved odorant-binding proteins participate in establishing tritrophic interactions. iScience, 25(7), Article 104664.

Productos recomendados: GAPDH comprado en abinScience 2022-07-01
F‑box and WD repeat‑containing protein 7 ameliorates angiotensin II‑induced myocardial hypertrophic injury via the mTOR‑mediated autophagy pathway

Liu Q, Han C, Wu X, Zhou J, Zang W. F-box and WD repeat-containing protein 7 ameliorates angiotensin II-induced myocardial hypertrophic injury via the mTOR-mediated autophagy pathway. Exp Ther Med. 2022;24(1):464. Published 2022 May 23. doi:10.3892/etm.2022.11391IF: 2.4 Q3

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